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Spasmolytic Polypeptide Expressing Metaplasia. Recent studies have demonstrated that spasmolytic polypeptide-expressing metaplasia SPEM in the mouse oxyntic mucosa arises from transdifferentiation of mature gastric chief cells. SPEM stands for Spasmolytic Polypeptide Expressing Metaplasia also Scanning Photoelectron Microscopy and 29 more What is the abbreviation for Spasmolytic Polypeptide Expressing Metaplasia. Spasmolytic polypeptide-expressing metaplasia SPEM is a regenerative lesion in the gastric mucosa and is a potential precursor to intestinal metaplasiagastric adenocarcinoma in a chronic inflammatory setting. The goal of these studies was to define the transcriptional changes associated with SPEM at the individual cell level in response to acute drug injury and chronic inflammatory damage in.
We explored the relationship between SPEM and IM in Epstein-Barr virus-associated EBVaGC and. Recently spasmolytic polypeptide-expressing metaplasia SPEM is proposed for pyloric gland-like metaplasia mainly in animal experiments. Spasmolytic polypeptidetrefoil factor 2expressing metaplasia SPEM is known to emerge after parietal cell loss and during Helicobacter pylori infection however its role in gastric ulcer repair is unknown. このSpasmolytic Polypetide Expressing metaplasia SPEM はヒト胃癌に隣接する胃粘膜やHelicobacter felis Hfelis を感染させたマウスラットの残胃癌モデルでも観察された. Spasmolytic polypeptide-expressing metaplasia SPEM is a regenerative lesion in the gastric mucosa and is a potential precursor to intestinal metaplasiagastric adenocarcinoma in a chronic inflammatory setting. Pylori infection and gastric adenocarcinoma.
Intestinal metaplasia and spasmolytic polypeptide-expressing metaplasia SPEM.
SPEM is a metaplastic mucous cell lineage with phenotypic characteristics of deep antral gland cells including strong expression of trefoil factor 2. The goal of these studies was to define the transcriptional changes associated with SPEM at the individual cell level in response to acute drug injury and chronic inflammatory damage in. SPEM is a metaplastic mucous cell lineage with phenotypic characteristics of deep antral gland cells including strong expression of Trefoil Factor 2 TFF2 previously designated as spasmolytic. In the presence of inflammation SPEM can advance into a more proliferative metaplasia with increased expression of. Recent studies have demonstrated that spasmolytic polypeptide-expressing metaplasia SPEM in the mouse oxyntic mucosa arises from transdifferentiation of mature gastric chief cells. Objective Spasmolytic polypeptide-expressing metaplasia SPEM is a regenerative lesion in the gastric mucosa and is a potential precursor to intestinal metaplasiagastric adenocarcinoma in a chronic inflammatory setting.